- decreased susceptibility to autoimmune diabetes / MGI
- periinsulitis / MGI
- decreased physiological sensitivity to xenobiotic / MGI
- decreased hematocrit / MGI
- abnormal liver physiology / MGI
- abnormal T cell differentiation / MGI
- abnormal cytokine secretion / MGI
- increased autoantibody level / MGI
- decreased susceptibility to parasitic infection / MGI
C.B-H2b/Orl
Status | Available to order |
EMMA ID | EM:00210 |
International strain name | C.B-H2b/Orl |
Alternative name | BALB/c.B-H2b/Cdta |
Strain type | Other |
Allele/Transgene symbol | H2b |
Gene/Transgene symbol | H2 |
Information from provider
Provider | CDTA unknown |
Provider affiliation | CNRS-Orleans |
Phenotypic information | The BALB/c.b mouse strain has a BALB/c genetic background but carries a different allele (H2b) derived from the BALB/c.B/Orl strain. The congenic mice are sensitive to increased immunodeficiency produced by murine leukemia viruses. |
References |
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Information from EMMA
Archiving centre | Institut de Transgenose, INTRAGENE, Orléans, France |
Disease and phenotype information
MGI phenotypes (allele matching)
MGI phenotypes (gene matching)
- decreased hematocrit / MGI
- abnormal liver physiology / MGI
- abnormal T cell differentiation / MGI
- increased susceptibility to bacterial infection / MGI
- abnormal immunoglobulin level / MGI
- increased IgM level / MGI
- abnormal cytokine secretion / MGI
- thyroid inflammation / MGI
- increased autoantibody level / MGI
- insulitis / MGI
- increased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to parasitic infection / MGI
- immune system phenotype / MGI
- periinsulitis / MGI
- abnormal T cell number / MGI
- decreased CD4-positive, alpha beta T cell number / MGI
- increased CD8-positive, alpha-beta T cell number / MGI
- decreased IgG1 level / MGI
- decreased IgG2a level / MGI
- increased physiological sensitivity to xenobiotic / MGI
- decreased physiological sensitivity to xenobiotic / MGI
- increased sensitivity to induced morbidity/mortality / MGI
Literature references
- Rapid development of murine AIDS is dependent of signals provided by CD54 and CD11a.;Makino M, Yoshimatsu K, Azuma M, Okada Y, Hitoshi Y, Yagita H, Takatsu K, Komuro K, ;1995;Journal of immunology (Baltimore, Md. : 1950);155;974-81; 7608573
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